潘氏细胞通过维生素D受体对抗致病菌入侵 | 热心肠日报

[IF:17.373]

Paneth cell alertness to pathogens maintained by vitamin D receptors

潘氏细胞通过维生素D受体维持对致病菌的警戒

10.1053/j.gastro.2020.11.015

11-17, Article

Abstract & Authors:展开

Abstract:收起
Background: Vitamin D exerts a regulatory role over mucosal immunity via the vitamin D receptor (VDR). While Paneth cells and their products are known to regulate the commensal and pathogenic microbiota, the role that VDRs in Paneth cells play in these responses is unknown.
Methods and Results: We identified the decreased intestinal VDR significantly correlated with reduction of an IBD risk gene ATG16L1 and Paneth cell Lysozymes in patients with Crohn’s Disease. We generated a Paneth cell specific VDR knockout (VDRΔPC) mice to investigate the molecular mechanisms. Lysozymes in the Paneth cells were significantly decreased in the VDRΔPC mice. Isolated VDRΔPC Paneth cells exhibited weakened inhibition of pathogenic bacterial growth and displayed reduced autophagic responses. VDRΔPC mice had significantly higher inflammation after Salmonella infections. VDRΔPC mice also showed high susceptibility to small intestinal injury induced by indomethacin, a nonsteroidal anti-inflammatory drug. Co-housing of VDRΔPC and VDRloxp mice made the VDRΔPC less vulnerable to the DSS-colitis, suggesting the transmission of protective bacterial from the VDRloxp mice. Thus, a lack of VDR in Paneth cells leads to impaired anti-bacterial activities and consequently increased inflammatory responses. Genetically and environmentally regulated VDRs in the Paneth cells may set the threshold for the development of chronic inflammation, as observed in inflammatory bowel diseases.
Conclusion: We provide new insights into the tissue-specific functions of VDRs in maintaining the Paneth cell alertness to pathogens in intestinal disorders. Targeting the VDR affects multiple downstream events within Paneth cells that inhibit intestinal inflammation and establish host defense against enteropathogens.

First Authors:
Rong Lu

Correspondence Authors:
Jun Sun

All Authors:
Rong Lu,Yongguo Zhang,Yinglin Xia,Jilei Zhang,Arthur Kaser,Richard Blumberg,Jun Sun

(0)

相关推荐

  • 细胞外微小囊泡介导功能失调脂肪细胞与心肌细胞之间的病理通讯是加重糖尿病小鼠缺血再灌注损伤的新机制

        本公众号每天分享一篇最新一期Anesthesia & Analgesia等SCI杂志的摘要翻译,敬请关注并提出宝贵意见 细胞外微小囊泡介导功能失调脂肪细胞与心肌细胞之间的病理通讯是加重 ...

  • 七氟醚可引起新生小鼠发育期大脑记忆功能损伤

    祝贺贵州医科大学麻醉学院15级科学学位研究生郭唯真顺利毕业 研究方向:机械通气肺损伤  指导老师:高鸿教授 高巨教授 研究生期间共发表文章3篇,其中北图核心2篇,参与国家自然科学基金项目1项     ...

  • 六个样本的单细胞数据如何发一篇10分以上的文章

    文章信息 清华大学陈晔光和杨雪瑞课题组及北京大学第三医院付卫课题组一起合作,于2020年2月3日在Journal of Experimental Medicine 杂志上在线发表了题为Single-c ...

  • 超实用干货帖 | 微生物领域选刊指南之四:肠道菌群选刊指南

    本文由微科盟中西科研君根据实践经验而整理,希望对大家有帮助. 微科盟原创微文,欢迎转发转载. 肠道微生物是人的第二大脑,近年来,肠道微生物的研究成为生命科学与医学研究的热门领域.前几次我们汇总了微生物 ...

  • 空间记忆相关蛋白的蛋白质组学研究

    C57BL/6J小鼠在评价空间记忆的多T迷宫(MTM)和Barnes迷宫(BM)中表现不同, 本研究拟寻找导致这种行为差异的海马蛋白. 应用双向凝胶电泳结合质谱鉴定和数据库检索, 分析比较经两种迷宫训 ...

  • 潘氏细胞

    潘氏细胞位于肠腺基底部 协助诊断溃疡性结肠炎.胃黏膜肠上皮化生等 Lendrum荧光桃红酒石黄法 试剂: 固定液:中性甲醛或Helly液 荧光桃红液:荧光桃红(Phloxine)B 0.5克,氯化钙( ...

  • 改变教科书:肠道效应T细胞主要由微生物塑造,而非传统范式 | 热心肠日报

    今天是第1730期日报. Nature 子刊:微生物时刻塑造着肠道效应T细胞! Nature Immunology[IF:20.479] ① 对SPF及无菌小鼠,或用不同表型偏向性的菌干扰的小鼠的肠道 ...

  • 中国道家潘氏静功_____双半合一大周天

    本功法共分三节,每节呼吸10次,如需延长练功时间,可以在每节中增加呼吸次数. 特点同时修炼上丹田(性宫).中丹田下丹田(命宫),即性命双修,气感强,可激发人体的各种潜能,开发特异功能:开天目(人体 ...

  • 厚仁村:潘氏家庙古风存

    厚仁村:潘氏家庙古风存     厚仁村位于兰江街道办事处驻地西8500米.村民委员会驻厚仁得名.地属丘陵,区域面积2.39平方公里,耕地1131亩.水面260亩.辖厚仁.下戴2个自然村,17个村民小组 ...

  • 荥阳白露源潘氏阳基图小序

    <荥阳白露源潘氏阳基图小序>: 盖闻氏族繁昌,必藉山川秀逸,子孙繁衍,端回祖考阴功.惟我德四公迹出名门,身居华胄,受露源之幽雅,恭甘水之清涟,匪苦嚣而苦隘,爰卜世而卜年,其东则山号毛桐,万 ...

  • 荥阳潘氏白露源潘村阳基小记

    <荥阳潘氏白露源潘村阳基小记>: 从来氏族之发祥由世德而山川秀逸亦居其半焉.我祖德四公,本光禄大卿公之余裔,大理寺少卿公之嫡礽簪缨累叶,代有闻人,乃厌桃坞之尘嚣,爱露源之幽雅,登高旁眺而遂 ...

  • 潘氏家训

    潘氏家训 1.敦孝弟 敦孝弟,凡后生见祖父与长辈老者,必须站立于傍,有问即答:有令即从.毋得踞傲自尊与厉声冲撞:酗酒放肆:恃逞才能:目无尊长者,责罚不恕. 2.尚廉耻 尚廉耻 ,为人必须端正.端正行事 ...

  • 潘氏钦定四库全书

    潘[荥阳羽音周文王毕公之子孙食采于潘因氏周] 潘崇[楚穆王立以其为太子之室与潘崇使为太师且掌环列之尹] 潘党[字叔党楚大夫与养由基蹲甲而射彻七札焉以示楚王曰君有二臣如此何忧于战]潘尫[字师叔楚大夫庄王 ...

  • 脑脊液潘氏(Pandy)试验

    [标本采集] 作腰椎穿刺后,抽取脑脊液2-5ml,置于清洁干燥的试管内. [正常值范围] 阴性. [临床意义] 正常脑脊液含有极微量的蛋白质,其中以白蛋白为主,潘氏试验为阴性反应.化脓性脑膜炎,结核性 ...